High glucose distinctively regulates Ca2+ influx in cytotoxic T lymphocytes upon target recognition and thapsigargin stimulation
dc.bibliographicCitation.firstPage | 2095 | eng |
dc.bibliographicCitation.issue | 12 | eng |
dc.bibliographicCitation.journalTitle | European Journal of Immunology | eng |
dc.bibliographicCitation.lastPage | 2098 | eng |
dc.bibliographicCitation.volume | 50 | eng |
dc.contributor.author | Zou, Huajiao | |
dc.contributor.author | Yang, Wenjuan | |
dc.contributor.author | Schwär, Gertrud | |
dc.contributor.author | Zhao, Renping | |
dc.contributor.author | Alansary, Dalia | |
dc.contributor.author | Yin, Deling | |
dc.contributor.author | Schwarz, Eva C. | |
dc.contributor.author | Niemeyer, Barbara A. | |
dc.contributor.author | Qu, Bin | |
dc.date.accessioned | 2020-12-29T09:41:53Z | |
dc.date.available | 2020-12-29T09:41:53Z | |
dc.date.issued | 2020 | |
dc.description.abstract | In CTLs: High glucose‐culture enhances thapsigargin‐induced SOCE but decreases target recognition‐induced Ca2+ influx. High glucose‐culture regulates expression of ORAIs and STIMs without affecting glucose uptake. More high glucose‐cultured CTLs are prone to necrosis after execution of killing. | eng |
dc.description.version | publishedVersion | eng |
dc.identifier.uri | https://doi.org/10.34657/4664 | |
dc.identifier.uri | https://oa.tib.eu/renate/handle/123456789/6035 | |
dc.language.iso | eng | eng |
dc.publisher | Hoboken, NJ : Wiley | eng |
dc.relation.doi | https://doi.org/10.1002/eji.202048577 | |
dc.rights.license | CC BY-NC 4.0 Unported | eng |
dc.rights.uri | https://creativecommons.org/licenses/by-nc/4.0/ | eng |
dc.subject.ddc | 570 | eng |
dc.subject.other | cytotoxic T lymphocytes | eng |
dc.subject.other | glucose | eng |
dc.subject.other | killing process | eng |
dc.title | High glucose distinctively regulates Ca2+ influx in cytotoxic T lymphocytes upon target recognition and thapsigargin stimulation | eng |
dc.type | Article | eng |
dc.type | Text | eng |
tib.accessRights | openAccess | eng |
wgl.contributor | INM | eng |
wgl.subject | Biowissensschaften/Biologie | eng |
wgl.type | Zeitschriftenartikel | eng |
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